See http://liveherpesvaccine.com/category/herpes-immunology/ http://liveherpesvaccine.com/category/herpes-immunology/ for a long-running detailed blog by a diff

See http://liveherpesvaccine.com/category/herpes-immunology/ http://liveherpesvaccine.com/category/herpes-immunology/ for a long-running detailed blog by a different scientist who has been working on similar ideas for years. He has shown that a mutated HSV virus which is not capable of causing clinical disease (but which does cause a lifelong infection by the "harmless" version) protects against the dangerous version. This new research seems to be in the same vein: > When introduced to a mouse, HSV-2 was able to use the HSV-1 gD to enter the mouse’s cells. Once inside, HSV-2 replicated abundantly, but because it could not produce gD, future progeny were unable to infect new cells. According to Herold, infected cells then became “little factories for making viral proteins” that spurred the immune system to produce antibodies to HSV-2. That sounds like some cells are infected with the mutated virus and this remains as a lifelong infection. So far there seems little hope that the FDA would approve such an approach, based on Halford's experience. (See linked blog.)