I'm not optimistic at all. It cured this one type of baldness because the baldness was a symptom of the underlying autoimmunity. If MPB were a classical autoim

I'm not optimistic at all. It cured this one type of baldness because the baldness was a symptom of the underlying autoimmunity. If MPB were a classical autoimmunity, it would have already been "cured" (as in, an effective treatment protocol would be laid out). Instead, it seems to be some not so subtle side effect of hormonal nuclear war, and scientists have spend decades hunting down the various little side chains to the main problem. The best hope yet for MPB seems to be PGD2 formation inhibitors or GPR44 antagonists which are still undergoing characterization. But my bet is that like DHT, PGD2 overexpression in scalps is simply yet another side effect of the greater convoluted underlying process, and that treating it directly won't be as effective as promised. (Or worse; GPR44 is implicated in some kinds of asthma as well, which means that a drug that targets the receptor might have a wider spectrum of activity -> worse side effects.)