LSD is primarily a 5HT2 agonist which leads to a decrease in dopamine pathways. This decrease in dopamine leads to taking it mimicing the "negative" symptoms of

LSD is primarily a 5HT2 agonist which leads to a decrease in dopamine pathways. This decrease in dopamine leads to taking it mimicing the "negative" symptoms of schizophrenia. this effect has been used in animal models to support various dopamine theories for us to learn more about schizophrenia, such as one of the current theories advocating for an increase in dopamine in mesolimbic brain pathway and decrease of dopamine in mesocortical pathways. So the drug is useful for inducing the symptoms, however once the drug is gone so are the symptoms. Schizophrenics have problems at some cellular level, be it receptor, transcription, translation, protein modification, etc that leads to their symptoms and the end effects that are similar to LSD. LSD causes the final step in the pathway, not the initial ones. Likewise for your drugs, you named two dopamine blockers (one selective for D_2 and one not). these treat schizophrenia in the sense that they minimise symptoms since they are working at the final step in what is wrong with the patients. They are not "cures" because they don't address the initial issue, which we hopefully will discover.