3 ms·

I don't exactly want to dispute that, but I would like to remark it's not entirely clear-cut. SARS-CoV-2 response could be as rapid as it was in no small part

by etiam 2y ago

I don't exactly want to dispute that, but I would like to remark it's not entirely clear-cut. SARS-CoV-2 response could be as rapid as it was in no small part because two decades of research on SARS-CoV more or less was applicable, and looking at genetic similarity alone contemporary variants of SARS-CoV-2 are now more different from the original strain isolated in Wuhan than that strain is different from SARS-CoV. So was the threat from pathogenic Coronaviruses really novel? To a degree, and depending on what aspects one chooses to emphasize maybe. Also, I'd say the pathogen itself doesn't necessarily need to do the changing in the context of emerging diseases. I'd take it to mean they have potential to become more prevalent or have a more significant impact, but that could easily also be due to changes in conditions, including human behavior.

JumpCrisscross 2y ago
> I would like to remark it's not entirely clear-cut Are you saying this as an epidimeologist? (I'm not an epidimeologist. I've only studied the math.) > looking at genetic similarity alone Epidimeology is the study of disease in a population. You can't look at the virus alone. The dividing line between emerging and novel is brighter than between emerging and existing. It has to do with whether the population has pre-existing immunity. SARS-CoV infection conferred no immunity to SARS-CoV-2. Getting infected with the Alpha variant, on the other hand, conferred immunity to Omega. > I'd say the pathogen itself doesn't necessarily need to do the changing in the context of emerging diseases. I'd take it to mean they have potential to become more prevalent or have a more significant impact, but that could easily also be due to changes in conditions, including human behavior Literally in the Wikipedia. "EIDs may also result from spread of an existing disease to a new population in a different geographic region, as occurs with West Nile fever outbreaks."

by etiam 2y ago

JumpCrisscross 2y ago

> I would like to remark it's not entirely clear-cut Are you saying this as an epidimeologist? (I'm not an epidimeologist. I've only studied the math.) > looking at genetic similarity alone Epidimeology is the study of disease in a population. You can't look at the virus alone. The dividing line between emerging and novel is brighter than between emerging and existing. It has to do with whether the population has pre-existing immunity. SARS-CoV infection conferred no immunity to SARS-CoV-2. Getting infected with the Alpha variant, on the other hand, conferred immunity to Omega. > I'd say the pathogen itself doesn't necessarily need to do the changing in the context of emerging diseases. I'd take it to mean they have potential to become more prevalent or have a more significant impact, but that could easily also be due to changes in conditions, including human behavior Literally in the Wikipedia. "EIDs may also result from spread of an existing disease to a new population in a different geographic region, as occurs with West Nile fever outbreaks."