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There’s no evidence this has anything to do with carbohydrates. Why would you think carbs have anything to do with an increased risk of Type 1 diabetes?

darkhorn 2y ago
Diabet is related with obesity and obesity is caused by consuming lots of carbonhydrates.
- stranded22 2y ago
  Type 1 diabetes is NOT related to obesity AT ALL. as per the article - T1D causation is a mixture of genetics and unknown factors. T1D and T2D are two separate conditions that share a similar name.
  - InMice 2y ago
    > T1D and T2D are two separate conditions that share a similar name. Not exactly. As you probably know, Type 1 diabetes involves no insulin production. Type 2 involves extreme, excessive insulin production. But at some point for some type 2 diabetics their pancreas can actually become so overworked it "gives out" and becomes incapable of producing insulin. They then actually become Type 1 diabetics and require insulin replacement to survive. Likewise, a doctor can prescribe insulin to a type 2 diabetic who's pancreas is still working (and without even ever testing their insulin responses) - which some argue should never be done because it only makes the insulin problem worse. Type 2 diabetes is completely reversible for probably most people via diet and lifestyle changes. But in the "chronic management" model of care extreme type 2 diabetics run the risk of becoming type 1 diabetics by being prescribed supplemental insulin.
    - stranded22 2y ago
      Thanks. T2D involves extreme excessive insulin production? That would surely put the person into hypoglycaemia and losing significant weight whereas the stereotypical T2D is hyperglycaemia, and overweight. I was under the impression that it is the opposite, that the pancreas can’t produce enough insulin (or the body resists it) and so glucose levels are difficult to get down. Metformin tablets slows down the glucose levels being pushed out by the liver. If that doesn’t work, then others try to increase insulin sensitivity.
- kleiba 2y ago
  > obesity is caused by consuming lots of carbonhydrates Sorry, what? Obesity is caused by taking in more calories than you burn off. One way to do that is through carbs, but you can just as easily consume too many calories by, say, eating to much fat. As a matter of fact, fat contains more calories per gram than carbs, although it's true that carbs are usually easier to eat in bulk before becoming satiated. Btw, bananas are actually relatively high in carbohydrates.
  - InMice 2y ago
    A strict view of calories in, out is silly. I see it repeated here over and over. In a way it's true - if you take in less than you "burn", you will eventaully starve and die. But really your hormonal response to food cannot be represented so simply. Think about it more, why can a type 1 diabetic eat thousands and thousands of calories in a day but still lose weight? They can take in 5000+ calories, they lose weight. Type 1 diabetics without insulin replacement will waste away no matter what they eat sitting around all day. Whats going on with the "simple" in/out theory here? On the flip side, someone with insulin resistence can eat a bunch of oats and will have insulin response that takes triple the time for insulin to fall back to baseline compared to a healthy person without resistance. They will also produce 2 or 3 times as much insulin compared to whats considered a healthy response. And their fasting insulin could also be elevated. And they will be hungrier, not satiated. And they gain weight no matter how much they try to "move more" and "burn it off". Insulin in this effect is a storage hormone, a growth hormone. Insulin prevents your body from making ketones bodies from your body fat ("burning fat"). It makes sense - if food is coming in we don't need to access our fat stores, we need to use the new food for energy to move around or new storage or thermogenesis. and get the incoming glucose into cells and out of the blood. Fat triggers an insulin response that is a fraction of what proteins or carbs would. Insulin above a fasting baseline causes an anabolic state in the body. Insulin at a low fasting base line causes a catabolic state. Fat has more calories per gram but will not spike someone's insulin and cause it to remain excessively and chronically elevated. Anytime insulin is elevated you cannot burn your fat stores. Carbohydrates are more likely to cause increased hunger and energy storage. Fat will not have this same effect. The rise in obesity runs in lock step with the history of the USDA guidelines declaring your energy intake should be 40-60% carbohydrates (hundreds of grams of carbs per day), the food pyramid, long term decreased fat consumption per capita, and the rise of ultra processed foods. Ultra processed foods that are: designed to be addictive, loaded with refined carbohydrates and/or added sugars, absent of fiber, and have a long shelf life for transportation and shelf storage. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6082688/ https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6082688/

darkhorn 2y ago

Diabet is related with obesity and obesity is caused by consuming lots of carbonhydrates.

stranded22 2y ago
Type 1 diabetes is NOT related to obesity AT ALL. as per the article - T1D causation is a mixture of genetics and unknown factors. T1D and T2D are two separate conditions that share a similar name.
- InMice 2y ago
  > T1D and T2D are two separate conditions that share a similar name. Not exactly. As you probably know, Type 1 diabetes involves no insulin production. Type 2 involves extreme, excessive insulin production. But at some point for some type 2 diabetics their pancreas can actually become so overworked it "gives out" and becomes incapable of producing insulin. They then actually become Type 1 diabetics and require insulin replacement to survive. Likewise, a doctor can prescribe insulin to a type 2 diabetic who's pancreas is still working (and without even ever testing their insulin responses) - which some argue should never be done because it only makes the insulin problem worse. Type 2 diabetes is completely reversible for probably most people via diet and lifestyle changes. But in the "chronic management" model of care extreme type 2 diabetics run the risk of becoming type 1 diabetics by being prescribed supplemental insulin.
  - stranded22 2y ago
    Thanks. T2D involves extreme excessive insulin production? That would surely put the person into hypoglycaemia and losing significant weight whereas the stereotypical T2D is hyperglycaemia, and overweight. I was under the impression that it is the opposite, that the pancreas can’t produce enough insulin (or the body resists it) and so glucose levels are difficult to get down. Metformin tablets slows down the glucose levels being pushed out by the liver. If that doesn’t work, then others try to increase insulin sensitivity.
kleiba 2y ago
> obesity is caused by consuming lots of carbonhydrates Sorry, what? Obesity is caused by taking in more calories than you burn off. One way to do that is through carbs, but you can just as easily consume too many calories by, say, eating to much fat. As a matter of fact, fat contains more calories per gram than carbs, although it's true that carbs are usually easier to eat in bulk before becoming satiated. Btw, bananas are actually relatively high in carbohydrates.
- InMice 2y ago
  A strict view of calories in, out is silly. I see it repeated here over and over. In a way it's true - if you take in less than you "burn", you will eventaully starve and die. But really your hormonal response to food cannot be represented so simply. Think about it more, why can a type 1 diabetic eat thousands and thousands of calories in a day but still lose weight? They can take in 5000+ calories, they lose weight. Type 1 diabetics without insulin replacement will waste away no matter what they eat sitting around all day. Whats going on with the "simple" in/out theory here? On the flip side, someone with insulin resistence can eat a bunch of oats and will have insulin response that takes triple the time for insulin to fall back to baseline compared to a healthy person without resistance. They will also produce 2 or 3 times as much insulin compared to whats considered a healthy response. And their fasting insulin could also be elevated. And they will be hungrier, not satiated. And they gain weight no matter how much they try to "move more" and "burn it off". Insulin in this effect is a storage hormone, a growth hormone. Insulin prevents your body from making ketones bodies from your body fat ("burning fat"). It makes sense - if food is coming in we don't need to access our fat stores, we need to use the new food for energy to move around or new storage or thermogenesis. and get the incoming glucose into cells and out of the blood. Fat triggers an insulin response that is a fraction of what proteins or carbs would. Insulin above a fasting baseline causes an anabolic state in the body. Insulin at a low fasting base line causes a catabolic state. Fat has more calories per gram but will not spike someone's insulin and cause it to remain excessively and chronically elevated. Anytime insulin is elevated you cannot burn your fat stores. Carbohydrates are more likely to cause increased hunger and energy storage. Fat will not have this same effect. The rise in obesity runs in lock step with the history of the USDA guidelines declaring your energy intake should be 40-60% carbohydrates (hundreds of grams of carbs per day), the food pyramid, long term decreased fat consumption per capita, and the rise of ultra processed foods. Ultra processed foods that are: designed to be addictive, loaded with refined carbohydrates and/or added sugars, absent of fiber, and have a long shelf life for transportation and shelf storage. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6082688/ https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6082688/