Apologies if this becomes a re-send! Overall-size heterogeneity could be partially epigenetically-driven right? I should try to read the paper, but I'm not a b

Apologies if this becomes a re-send! Overall-size heterogeneity could be partially epigenetically-driven right? I should try to read the paper, but I'm not a biologist. I've been listening to Michael Levin quite a bit though, and from what he says it seems like there's some very interesting ideas around how genes set up or affect 'vmem' (this intercellular voltage-pattern 'memory' of what-to-grow-into, that cells retain in a non-DNA-based way after the initial setup).. I think it is interesting to ponder what might be able to get through the gamete-gates and still affect the vmem pattern by going around the side (ie via cytoplasm/membranes or even encoded into the cytoskeleton?! Rather than just the information that goes via the nucleus).