Overall-size heterogeneity could be partially epigenetically-driven right? I should try to read the paper, but I'm not an academic biologist. I've been listenin

Overall-size heterogeneity could be partially epigenetically-driven right? I should try to read the paper, but I'm not an academic biologist. I've been listening to Michael Levin quite a bit though, and from what he says it seems like there's some very interesting ideas around how genes participate (or don't!) in setting up or affecting 'vmem' (that's what they call this intercellular voltage-pattern 'memory' of what-to-grow-into, that cells apparently retain in a non-DNA-based way after the initial setup).. I think it is interesting to ponder what might be able to get through the gamete-gates and still affect the vmem pattern (ie the morphological-target, which would usually probably include 'how large to make it' right?) by going around the side (ie via cytoplasm/membranes or even encoded into the cytoskeleton?! Rather than just what goes into the next generation via the sequence in the DNA in the nucleus).