This doesn't make sense. HIV virus is generally not "substrate limited"/competing with other virii in its propagation in patients in (temporary) remission -- t

This doesn't make sense. HIV virus is generally not "substrate limited"/competing with other virii in its propagation in patients in (temporary) remission -- that's basically all of them that don't have AIDS. HIV retro integrates into host DNA and sporadically resurges on a stochastic basis, the drug cocktails in use inhibits this process. Generally the viral load of a patient with HIV is miniscule but must always be kept in check. Note that the point of a crispr treatment is that it would delete dormant HIV sequences that are hiding in the host DNA and not currently producing virii but threatening to pop up at some time in the future. Obviously this too is subject to evolutionary pressures but almost certainly not the ones you are thinking of.