But lots of aspects and features of SARS-CoV2 point to it being a natural virus with a human specific furin cleavage site inserted into it. In fact it looks alm

But lots of aspects and features of SARS-CoV2 point to it being a natural virus with a human specific furin cleavage site inserted into it. In fact it looks almost exactly what you would expect from the type of experiments outlines in the DEFUSE Proposal https://www.documentcloud.org/documents/21066966-defuse-proposal https://www.documentcloud.org/documents/21066966-defuse-prop... The fact that the location happens to be in the exact location proposed in experiments and commonly used in virology, and the fact that SARS-CoV2 binds to Human ACE2 more efficiently than any other species really stands out: > Spike protein exhibited the highest binding to human (h)ACE2 of all the species tested. . . > These findings show that the earliest known SARS-CoV-2 isolates were surprisingly well adapted to bind strongly to human ACE2, helping explain its efficient human to human respiratory transmission https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8225877/ https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8225877/ > Our observations suggest that by the time SARS-CoV-2 was first detected in late 2019, it was already pre-adapted to human transmission to an extent similar to late epidemic SARS-CoV. However, no precursors or branches of evolution stemming from a less human-adapted SARS-CoV-2-like virus have been detected https://www.biorxiv.org/content/10.1101/2020.05.01.073262v1 https://www.biorxiv.org/content/10.1101/2020.05.01.073262v1 Not to mention that it's absurd to think that such an infectious virus would have been circulating in Raccoon Dogs or Civets and just simply disappear after the first human was infected with zero traces to be found. Like when humans passed covid to white tailed deer that did not stop the virus from circulating in humans. Are we supposed to just accept SARS-CoV2 spillover was the result of an "Immaculate Infection">?