Makes sense. I found this too in a science journal: > The exact pathophysiology of long COVID is unknown but is likely to be multifactorial, including the infl

Makes sense. I found this too in a science journal: > The exact pathophysiology of long COVID is unknown but is likely to be multifactorial, including the inflammatory cascade during acute infection and persistent viral replication. Mechanistic in-silico modelling predicts that translation of SARS-CoV-2 viral proteins is a particularly sensitive target for inhibition of viral replication, and previous studies have shown that metformin is capable of suppressing protein translation via mammalian target of rapamycin (mTOR) inhibition https://www.science.org/content/blog-post/long-covid-shows-card https://www.science.org/content/blog-post/long-covid-shows-c...