It's even worse. This study was done in worms. A species of worms widely known for having literally one of the most simple neural structures of any animal on th

It's even worse. This study was done in worms. A species of worms widely known for having literally one of the most simple neural structures of any animal on the planet (they have like 300 Neurons in total). Those worms can't even develop Parkinson's. So what they observed is that literally feeding the bacterium in large quantities to those worms coincides with buildup of a certain protein that regulates synaptic activity and whose build up is observed in a certain share of humans with Parkinson's. But the worms were also genetically modified to enhance their susceptibility towards that protein. So this result doesn't just have to jump the causality hoop, it first has to make the jump to independent confirmation. Because there is a lot of potential for unintended bias. Even if it manages that, it also has to make the jump to mammals and after that it could still fail in the clinical stage. It is at best news in a field deprived of breakthroughs for a long time, but it is far from a real breakthrough.