If it is not DHTs fault (and by proxy Ts, as more T = more DHT) then why do finasteride and dutasteride work so very well? At this point, rejecting as main cul

If it is not DHTs fault (and by proxy Ts, as more T = more DHT) then why do finasteride and dutasteride work so very well? At this point, rejecting as main culprit for AGA either too much DHT or a DHT sensitivity that is to high or, worst case, both at once in one man, is nothing but crazy fringe science. To stress this out, the vast majority of men will keep their hair forever with oral minoxodil and oral finasteride and someone not keeping it on oral dutasteride instead of finasteride is a freak of nature.