They mention in the article that it had a dose-dependent effect on renal function that was both predictable and reversible. You're definitely correct, in that I

They mention in the article that it had a dose-dependent effect on renal function that was both predictable and reversible. You're definitely correct, in that I wouldn't trust any fat-loss mechanism that works through accelerating apoptosis without extensive human research. However, it's effect on various cells is understood, as it is determined by the type of protein evident on the cell surface. Fat cells are understood(under the vascular mapping prepared by the lab in question) to have this in far greater numbers than any other cell type.