Not really. There is plenty of criticism of the amyloid hypothesis within the field, and even specifically of the oligomer variant thereof that Schrag focused

Not really. There is plenty of criticism of the amyloid hypothesis within the field, and even specifically of the oligomer variant thereof that Schrag focused on. You can even read that criticism in the link I provided. Schrag's investigation did not (in my opinion) discover any fraud in the Lesné paper, even if the paper perhaps over-states its case. The amyloid hypothesis has several very strong strands of evidence in its favor, specifically that it is (currently) the only way to reconcile the indisputable genetic and pathological evidence for mutations in the genes APP and presenilin both producing early-onset Alzheimer's disease. Amyloid is derived from the action of presenilin on APP. Until an alternate way of reconciling this observation is found, the amyloid hypothesis will always have supporters.