> they prescribe both boosters, and inhibitors, to treat the same symptoms So here's the interesting thing - there's evidence that 5-HT2a receptors are way too

> they prescribe both boosters, and inhibitors, to treat the same symptoms So here's the interesting thing - there's evidence that 5-HT2a receptors are way too dense in people with depression. Antagonists obviously have a blocking effect, but increasing serotonin at the synapse counterintuitively has a similar effect - your body responds to it by paradoxically downregulating 5-HT2a receptors. Two totally opposite approaches with a similar outcome.