> For sucrose, mice apparently have the opposite reaction that primates have in this study. Maybe the guys who did the linked study didn't read it. @stevenwoo

> For sucrose, mice apparently have the opposite reaction that primates have in this study. Maybe the guys who did the linked study didn't read it. @stevenwoo Did YOU read the study, because it seems as if you may have skipped the very beginning reading... This is a direct quote. > The association of glucose with survival is inverse for mice versus monkeys/humans. That is -not at all- what the study in this post is reviewing, and is not the conclusion you're purporting. The study you linked looks at how glucose and aging work together. The study in this post is looking to how glucose affects the brain function itself, and what inhibitors are triggered by high-sucrose in a diet. > he high-sucrose diet induced microcapillary impairments and reduced brain glucose uptake in brains of Glo1-deficient mice. Aspirin protected against this angiopathy, enhancing brain glucose uptake and preventing abnormal behavioral phenotypes. Similar vascular damage to our model mice was found in the brains of randomly collected schizophrenia and bipolar disorder patients, suggesting that psychiatric disorders are associated with angiopathy in the brain caused by various environmental stresses, including metabolic stress. That's a direct quote from this post's linked study. These two studies are dissimilar enough to be completely different in comparison and their conclusions are not similar because the studies look at different effects of sucrose. This post's linked study looks at immediacy and how the sucrose interacts with the brain. Your post looks at the effects of aging and possible links to high-sucrose and fasting diets.