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Basically with Type 2 diabetes it is runaway inflammation with a genetic makeup that causes the insulin receptors to get less efficient. Insulin is the key that

Basically with Type 2 diabetes it is runaway inflammation with a genetic makeup that causes the insulin receptors to get less efficient. Insulin is the key that allows the glucose to get transferred into a cell. Once the body is starving for carbohydrates it begins a process to break down fats and even protein into an energy source. This produces toxins, ketones. That is a brief overview of the biggest factors. To prevent, or reverse this downward spiral, eating better will help and there is a simple life hack. Melatonin upregulates insulin receptors, this works for everyone not just already diagnosed diabetics. With Type 1 autoimmune diabetes melatonin can reduce insulin requirements but won't eliminate the need for insulin. With Type 2 and with prediabetes melatonin along with minding your diet can have a dramatic effect. Increasing the ratio of unsaturated fats (health plant oils not palm oil) like olive oil and increasing plant based protein sources like eating beans, peas or lentils for some of your meals will help reduce the risk of diabetic complications. Another hack if you go with any amount of a plant based diet is to eat raw miso from the tub or to carefully make it into broth with water under 130 fahrenheit. Miso is full of microbes that make plant protein more digestible and useable by the body than any other source.

bpizzi 5y ago
> Once the body is starving for carbohydrates it begins a process to break down fats and even protein into an energy source. This produces toxins, ketones. You may want to read some (hopefully) unbiased information on that subject : https://en.wikipedia.org/wiki/Ketone_bodies https://en.wikipedia.org/wiki/Ketone_bodies https://en.wikipedia.org/wiki/Ketogenesis https://en.wikipedia.org/wiki/Ketogenesis For example: > Both [two of the three types of ketone bodies] are 4-carbon molecules that can readily be converted back into acetyl-CoA [which can directly fuel the krebs cycle] by most tissues of the body, with the notable exception of the liver. Which is not really matching the definition of 'toxins'. In my mind, saying that ketone bodies are toxins could be suggested in the fact that ketoacidosis is a real threat when a metabolism is affected by T1. But, again, T1 is not T2, and it's not the condition of a healthy metabolism, which is fully capable of handling ketone bodies, even thriving on it (glucose is fine, too, I'm not trying to sell that line).
- Projectiboga 5y ago
  Oh thanks, I've been T1 since 1982, so I'm not surprised that science has improved. Keto-acidosis is a toxic state for T1 diabetics at least, I'm not sure if T2 can handle them better.

bpizzi 5y ago

> Once the body is starving for carbohydrates it begins a process to break down fats and even protein into an energy source. This produces toxins, ketones. You may want to read some (hopefully) unbiased information on that subject : https://en.wikipedia.org/wiki/Ketone_bodies https://en.wikipedia.org/wiki/Ketone_bodies https://en.wikipedia.org/wiki/Ketogenesis https://en.wikipedia.org/wiki/Ketogenesis For example: > Both [two of the three types of ketone bodies] are 4-carbon molecules that can readily be converted back into acetyl-CoA [which can directly fuel the krebs cycle] by most tissues of the body, with the notable exception of the liver. Which is not really matching the definition of 'toxins'. In my mind, saying that ketone bodies are toxins could be suggested in the fact that ketoacidosis is a real threat when a metabolism is affected by T1. But, again, T1 is not T2, and it's not the condition of a healthy metabolism, which is fully capable of handling ketone bodies, even thriving on it (glucose is fine, too, I'm not trying to sell that line).

Projectiboga 5y ago
Oh thanks, I've been T1 since 1982, so I'm not surprised that science has improved. Keto-acidosis is a toxic state for T1 diabetics at least, I'm not sure if T2 can handle them better.