> It doesn't produce any alternative lines for thinking about treatment, causality, etc. The whole "genes that shouldn't be on being switched on" thing made me

> It doesn't produce any alternative lines for thinking about treatment, causality, etc. The whole "genes that shouldn't be on being switched on" thing made me think about David Sinclair's work on aging, and his information theory of aging. Has anyone been really looking into ways to avoid damaging (or ways to repair) epigenetic information as a potential solution to cancer?