No. >The problem sparks when a viral strain, normally happily living in a bat, pig, or rodent That's the rub right there. One of the reasons we were successf

No. >The problem sparks when a viral strain, normally happily living in a bat, pig, or rodent That's the rub right there. One of the reasons we were successful with smallpox is there wasn't an animal reservoir. One virus + no animal reservoirs + good vaccine = can be successful. Multiple known viruses and strains with multiple reservoirs and no good vaccines means this will not happen. More to the point, this isn't really what the paper was looking at all. The actual (OA) publication[1] identified common and conserved areas which could potentially be used to study and target viruses throughout out the family. This might help with human SARS-CoV2 therapeutics, but it's main benefit is showing how molecular analyses will help fight future coronaviruses. It does not ask or answer whether we can "engineer a universal vaccine against the entire viral family" nor did it "[draw] up a scientific recipe to potentially end coronaviruses once and for all." 1: https://science.sciencemag.org/content/early/2020/10/14/science.abe9403 https://science.sciencemag.org/content/early/2020/10/14/scie...