Not really, as the paper itself says. It's been documented for MERS and SARS. COVID-19 is special with its higher likelihood of such problems and presence of im

Not really, as the paper itself says. It's been documented for MERS and SARS. COVID-19 is special with its higher likelihood of such problems and presence of immune infiltration of the infected heart, which causes more problems than the above two. It's much less discriminating in the kind of ACE2 receptor it uses to enter cells, meaning it can infect many more organs effectively and also spread more effectively, including zoonotic spread. I'd like to see if general ACE inhibitors do anything to it.