It's true that the G614 form overtook the D614 form, but that is not necessarily because it's more infectious - it could be drift and fixation. The news and vi

It's true that the G614 form overtook the D614 form, but that is not necessarily because it's more infectious - it could be drift and fixation. The news and views bit on that Cell paper puts it in context: https://www.cell.com/cell/pdf/S0092-8674(20)30817-5.pdf https://www.cell.com/cell/pdf/S0092-8674(20)30817-5.pdf Another study didn't find an effect on transmission: https://www.biorxiv.org/content/10.1101/2020.05.21.108506v4 https://www.biorxiv.org/content/10.1101/2020.05.21.108506v4 > The D614G mutation displays only three independent emergences that qualify for inclusion in our analyses. While this limits our power to detect a statistically significant association with transmissibility, the low number of recurrent mutations leading to the D614G allele suggests that, rather than being a driver a of transmission itself, it arose early and went up in frequency by hitchhiking with one of the deepest branches in the global phylogeny as the SARS-CoV-2 population expanded. People are still working on this, and it should become clearer in time.