3 ms·

As long as we're tossing around citations to the scientific literature which supposedly support our arguments, here's one I'm sure you will enjoy: https://www.m

• __blockcipher__ 6y ago
  See, they use general human coronaviruses as a model, instead of SARS-1, which is incredibly functionally and structurally similar to SARS-2. Why would you willfully ignore the enormous research literature showing enduring immunity developing from SARS-1? Oh, right, because either you haven't read it or you don't like that it doesn't support your conclusions. It's like, imagine we're discussing H1N1 reinfection, and we have a highly similar H1N0 which varies very slightly, and we know that doesn't lead to reinfection. But instead you look at a number of Influenza viruses in the same family but not nearly as similar. Don't you see how ridiculous that is? We obviously should use SARS-1 as a model for SARS-2.