I'm not quite following how the relationship between microbiome science and an infection of the microbiome is minimal. But more to your point, using the mg dat

I'm not quite following how the relationship between microbiome science and an infection of the microbiome is minimal. But more to your point, using the mg data from their "healthy" population along with the open-label data in the literature seems like a good way to construct RCTs, no? Data from open label studies isn't speculative... it's just not phase 3. In case you're curious, here are two papers on UC: https://www.ncbi.nlm.nih.gov/pubmed/28214091 https://www.ncbi.nlm.nih.gov/pubmed/28214091 and https://www.ncbi.nlm.nih.gov/pubmed/29052237 https://www.ncbi.nlm.nih.gov/pubmed/29052237