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It's an indirect lie. Fat is what causes you to gain weight, and then excess calories. If your diet is not in excess of fat and calories then you can lose weigh

by spraak 8y ago

It's an indirect lie. Fat is what causes you to gain weight, and then excess calories. If your diet is not in excess of fat and calories then you can lose weight with sugar, but no, it doesn't have some magical weightloss chemical in it.

village-idiot 8y ago
That’s not quite right either. You need fat and sugar together. Humans can’t synthesize fat easily, so dietary fat is what gets stored. But insulin is the chemical responsible for storing said fat, and insulin levels are heavily modulated by sugar and other simple carbohydrates. The rub is that humans need some level of dietary fat. Without it we go into “rabbit starvation” and die. What we don’t actually need is carbohydrate, we can live just fine without it for arbitrary periods of time. Since any human in a non-extreme situation will have dietary fat and can control their carbs, it’s colloquially correct to say that “sugar causes weight gain”, even if the technical answer is more complicated.
- spraak 8y ago
  The human body is physiologically a complex (starch) and simple (sugar) carbohydrate machine. There is enough fat in rice or oats, for example, to survive. Yes the human body can live without carbohydrates, but it's basically in adaptive illness that it does. If you start talking about the Eskimo or Inuit, I'll point to their extremely short lifespan and epic numbers of heart disease.
will_brown 8y ago
>It's an indirect lie. Fat is what causes you to gain weight, and then excess calories. Sugar triggers insulin, insulin triggers storage of glucose in fat cells. There is a lot more to energy production/atp/Krebs cycle than calories in/calories out.
- CuriousSkeptic 8y ago
  Glucose triggers insulin, fructose does not. Yet fructose probably contributes more to insuline resistance long term than glucose does. The mechanism, as I understand it is that fructose essentially turns into fatty liver, which increases the insuline resistance, the increased IR in turn causes higher insulin levels from ingesting glucose at a later time causing higher median levels of insulin, also causing increased IR. Point being, the damage done from fructose isn’t measurable from the effects of a single meal. And the insulin raise from glucose is probably not that bad given a healthy start state.

by spraak 8y ago

village-idiot 8y ago

That’s not quite right either. You need fat and sugar together. Humans can’t synthesize fat easily, so dietary fat is what gets stored. But insulin is the chemical responsible for storing said fat, and insulin levels are heavily modulated by sugar and other simple carbohydrates. The rub is that humans need some level of dietary fat. Without it we go into “rabbit starvation” and die. What we don’t actually need is carbohydrate, we can live just fine without it for arbitrary periods of time. Since any human in a non-extreme situation will have dietary fat and can control their carbs, it’s colloquially correct to say that “sugar causes weight gain”, even if the technical answer is more complicated.

spraak 8y ago
The human body is physiologically a complex (starch) and simple (sugar) carbohydrate machine. There is enough fat in rice or oats, for example, to survive. Yes the human body can live without carbohydrates, but it's basically in adaptive illness that it does. If you start talking about the Eskimo or Inuit, I'll point to their extremely short lifespan and epic numbers of heart disease.

will_brown 8y ago

>It's an indirect lie. Fat is what causes you to gain weight, and then excess calories. Sugar triggers insulin, insulin triggers storage of glucose in fat cells. There is a lot more to energy production/atp/Krebs cycle than calories in/calories out.

CuriousSkeptic 8y ago
Glucose triggers insulin, fructose does not. Yet fructose probably contributes more to insuline resistance long term than glucose does. The mechanism, as I understand it is that fructose essentially turns into fatty liver, which increases the insuline resistance, the increased IR in turn causes higher insulin levels from ingesting glucose at a later time causing higher median levels of insulin, also causing increased IR. Point being, the damage done from fructose isn’t measurable from the effects of a single meal. And the insulin raise from glucose is probably not that bad given a healthy start state.