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"Cholesterol levels" is a blunt term to use, there are HDL, LDL (with different particle sizes) etc. Correlation != causation "The total body of evidence sugge
by padwan 9y ago
"Cholesterol levels" is a blunt term to use, there are HDL, LDL (with different particle sizes) etc.
Correlation != causation
"The total body of evidence suggests that attention should
be shifted from the harmful effects of dietary SAFA per
se, to the prevention of the accumulation of SAFA in
body lipids. This shift would emphasise the importance
of reducing dietary CHO, especially CHO with a high
glycaemic index, rather than reducing dietary SAFA"
http://www.njmonline.nl/getpdf.php?id=1095 http://www.njmonline.nl/getpdf.php?id=1095
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"A meta-analysis of prospective epidemiologic studies showed that there is no significant evidence for concluding that dietary saturated fat is associated with an increased risk of CHD or CVD"
http://ajcn.nutrition.org/content/early/2010/01/13/ajcn.2009.27725.abstract http://ajcn.nutrition.org/content/early/2010/01/13/ajcn.2009...
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"High intake of cholesterol results in less atherogenic low-density lipoprotein particles in men and women independent of response classification"
http://www.ncbi.nlm.nih.gov/pubmed/15164336 http://www.ncbi.nlm.nih.gov/pubmed/15164336
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"A high-fat, high–saturated fat diet is associated with diminished coronary artery disease progression in women with the metabolic syndrome."
http://ajcn.nutrition.org/content/80/5/1102.full http://ajcn.nutrition.org/content/80/5/1102.full
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https://denversdietdoctor.com/lchf-15-years-coronary-artery-calcium-score-zero/ https://denversdietdoctor.com/lchf-15-years-coronary-artery-...
- cryptokush 9y agoCan you please explain why in direct feeding metabolic ward studies we see a direct link between saturated fat intake and LDL cholesterol levels? We fully understand the mechanisms behind this. Epidemiologic studies of sick populations are useless. http://www.bmj.com/content/314/7074/112 http://www.bmj.com/content/314/7074/112