3 ms·
Maybe. It’s really too early to say. Even assuming the mechanism described here is correct, there’s a difference between mice and humans, and there’s a differen
by klmr 9y ago
Maybe. It’s really too early to say. Even assuming the mechanism described here is correct, there’s a difference between mice and humans, and there’s a difference between merely being stressed and a systemic stress response that would affect the sperm production (which, by the way, doesn’t occur just during sex but before).
And, finally, the effect on the offspring is completely unclear. One of the effects, which was reported here, is that offspring is less stressed (why? how? we don’t know; that’s a major missing puzzle piece). But systemic stress has quite a few, and partially drastic, effects — increasing expression of a particular microRNA is just one of many consequences. It also generally negatively affects semen quality (e.g. [1]), which doesn’t fare well for procreation.
And even just that particular microRNA in question, MiR-206, is tentatively implicated in multiple disease predispositions. You might make your children slightly less stressed. Or you might make it slightly more (or less) likely for them to acquire muscular dystrophy or schizophrenia.
[1] https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4382866/ https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4382866/
- mygo 9y ago>and there’s a difference between merely being stressed and a systemic stress response that would affect the sperm production (which, by the way, doesn’t occur just during sex but before). According to the way the research was described, this mechanism acts outside of the genetic code, so it isn’t a matter of when sperm is produced. These particles can latch on to the gamete long after the gamete is produced. For example they can be present along the lining or interstitial fluids in the epididymis, where sperm passes through from the testes during reproduction.