Basicly just different severities. Afaik no clear indications that they have different causes, so I don't understand the reason for OPs objection. I would assum

Basicly just different severities. Afaik no clear indications that they have different causes, so I don't understand the reason for OPs objection. I would assume their results are spanning all types, or it would have been spesifically mentioned. Most research seems to be done on the more severe type, tho. > They put Acinetobacter and Akkermansia bacteria into samples of healthy blood. The microbes increased the kind of cells that activate immune attack (they’re called T helper cells) and decreased the kind (regulatory T cells) that suppress excessive immune responses, including autoimmune attacks as in MS. As far as I understand the article it seems like they have replicated the effect MS has on the T-cells, and that is not subtype spesific. If they could inhibit this effect it would be a fix across all subtypes, even if it doesn't fix the underlying problem.