Neoplastic TKs are not on/off switches; they are always on. Once you inhibit effectively, they are always off, like you said. But you still have a copy of the n

Neoplastic TKs are not on/off switches; they are always on. Once you inhibit effectively, they are always off, like you said. But you still have a copy of the normal TK, so most likely things could be perfectly normal if you identified a highly specific TK inhibitor. The current -inibs just aren't specific enough yet, so they have serious side effects. I argue that antibiotics get to the heart of an infection, not that they only kill the bacteria you want to kill. Is it possible to create a drug or engineer a phage to do wo? Yes, sure. But the cost tradeoff is not worth it, on balance. Very few people develop any side effects from most antibiotic treatment, much less weeks of GI discomfort. It definitely happens, but you outlined the treatment for it -and eating yogurt is much cheaper than paying for a theoretical, patented, new superantibiotic that causes mo GI problems.