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> Sugar (or bad food) is an addiction. It's not about nutritionists or scientific evidence, it's about having enough mental strength to stay away from bad foods

> Sugar (or bad food) is an addiction. It's not about nutritionists or scientific evidence, it's about having enough mental strength to stay away from bad foods. You're close. Attempting to do so while not having any understanding of why things happen is a recipe for disaster. It must be reasoned that obesity is a disorder caused by something more then just 'poor eating habits'. There are physiotypes that will simply resist obesity on the worst of diets. I don't need to link a paper for this, this should be common sense from anyone's experience around enough people. So, if you reason that obesity is a disorder or sickness... you would then try to treat the cause, not the symptom. In the same sense that a person with a peanut allergy would simply avoid peanuts, a person with obesity may have this state caused by disturbed leptin/insulin signaling. A diet must then be designed to compensate for that disorder. (we know that once surplus white adipose tissue is grown, there's no going back, a modified diet must be adapted for life or those buckets just start filling again)

everial 10y ago
> we know that once surplus white adipose tissue is grown, there's no going back, a modified diet must be adapted for life or those buckets just start filling again Mind providing more references/reliable pointers on this?
- joecool1029 10y ago
  Apologies, I can't find the precise paper covering what I mean. You will find that there's a suspicious lack of studies on formerly obese people. Paper to get you thinking on how WAT acts on signaling: http://www.cambridge.org.secure.sci-hub.bz/core/journals/proceedings-of-the-nutrition-society/article/div-classtitlephysiological-role-of-adipose-tissue-white-adipose-tissue-as-an-endocrine-and-secretory-organdiv/6F914782A23DBDEC3E582833F6A16717 http://www.cambridge.org.secure.sci-hub.bz/core/journals/pro... Paper on WAT quantity and its effects on metabolic syndrome. http://onlinelibrary.wiley.com/doi/10.1111/eci.12519/pdf http://onlinelibrary.wiley.com/doi/10.1111/eci.12519/pdf . Where this one gets me thinking is that for some people, growing WAT may actually be an adaptation to prevent metabolic disorder from a poor diet. What we know is WAT can hold about 4x its size in fat before it divides into more. What we don't really know for humans is whether long term weight loss allows us to lose some of these, or whether they just deflate.... some rat studies show there can be a reduction, but for humans it would require repeated biopsy......
  - everial 10y ago
    Thanks for the paper links.
  - HeavenBanned 10y ago
    > There are physiotypes that will simply resist obesity on the worst of diets. Apologies for misunderstanding, but are you saying here that some individuals are able to eat as much as, say, a 300 lb obese male, yet somehow weigh-in at a rather fit (for example) 160 lb due to _genetic composition_?

everial 10y ago

> we know that once surplus white adipose tissue is grown, there's no going back, a modified diet must be adapted for life or those buckets just start filling again Mind providing more references/reliable pointers on this?

joecool1029 10y ago
Apologies, I can't find the precise paper covering what I mean. You will find that there's a suspicious lack of studies on formerly obese people. Paper to get you thinking on how WAT acts on signaling: http://www.cambridge.org.secure.sci-hub.bz/core/journals/proceedings-of-the-nutrition-society/article/div-classtitlephysiological-role-of-adipose-tissue-white-adipose-tissue-as-an-endocrine-and-secretory-organdiv/6F914782A23DBDEC3E582833F6A16717 http://www.cambridge.org.secure.sci-hub.bz/core/journals/pro... Paper on WAT quantity and its effects on metabolic syndrome. http://onlinelibrary.wiley.com/doi/10.1111/eci.12519/pdf http://onlinelibrary.wiley.com/doi/10.1111/eci.12519/pdf . Where this one gets me thinking is that for some people, growing WAT may actually be an adaptation to prevent metabolic disorder from a poor diet. What we know is WAT can hold about 4x its size in fat before it divides into more. What we don't really know for humans is whether long term weight loss allows us to lose some of these, or whether they just deflate.... some rat studies show there can be a reduction, but for humans it would require repeated biopsy......
- everial 10y ago
  Thanks for the paper links.
- HeavenBanned 10y ago
  > There are physiotypes that will simply resist obesity on the worst of diets. Apologies for misunderstanding, but are you saying here that some individuals are able to eat as much as, say, a 300 lb obese male, yet somehow weigh-in at a rather fit (for example) 160 lb due to _genetic composition_?