Here is an article that talks about possible mechanisms: http://sci-hub.io/http://content.iospress.com/articles/journal-of-alzheimers-disease/jad150290 http://s

Here is an article that talks about possible mechanisms: http://sci-hub.io/http://content.iospress.com/articles/journal-of-alzheimers-disease/jad150290 http://sci-hub.io/http://content.iospress.com/articles/journ... "Another implication of the ability of cholinergic antagonists to promote dementing diseases is that sufficient choline needs to be provided so that adequate PC production can continue. This might most effectively be done by administering the choline as a component of a nutrient mixture that contains all three of the PC precursors which limit PC’s rate of synthesis: uridine as its monophosphate; DHA or EPA; and choline [24]. This mixture has been shown to diminish Aβ formation in experimental animals [32], and could have the added benefit of partially restoring the deficient brain synapses."